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Short-term intracoronary administration of testosterone induces coronary artery dilation and increases coronary blood flow in men with established coronary artery disease . Therefore, an interplay of endogenous factors of the patient’s risk profile in addition to the testosterone dosing may be issues of whether or not opposed occasions happen. Moreover, testosterone has been used in the therapy of decreased muscle mass and bone density in addition to anemia. The role testosterone might play in the improvement of cardiovascular disease has more and more become the topic of present interest .
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In addition, this patient also had polycythemia, likely secondary to high-dose testosterone. The affected person underwent successful percutaneous intervention of the proper coronary artery. Physicians and sufferers should concentrate on the clinical consequences of anabolic steroid abuse. Erythrocytosis has been reported as a possible impact of testosterone, which could be beneficial in patients with anemia.
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Direct myocardial cell damage may occur by disruption of mitochondria and induction of intrafibrillar collagen dysplasia (Melchert and Welder 1995; Sullivan et al 1998). Cellular injury ends in fibrosis, which is normally a potential nidus for ventricular arrhythmias. Patients generally develop left ventricular hypertrophy, which can be related to hypertension, one other opposed effect of AAS. The left anterior artery demonstrates a 95% stenosis after the first septal branch. Scientific and technological companies and analysis and design relating thereto; industrial analysis and analysis companies; design and development of pc hardware and software program.
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